Endoplasmic reticulum stress drives proteinuria-induced kidney lesions via Lipocalin 2

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Endoplasmic reticulum stress drives proteinuria-induced kidney lesions via Lipocalin 2

In chronic kidney disease (CKD), proteinuria results in severe tubulointerstitial lesions, which ultimately lead to end-stage renal disease. Here we identify 4-phenylbutyric acid (PBA), a chemical chaperone already used in humans, as a novel therapeutic strategy capable to counteract the toxic effect of proteinuria. Mechanistically, we show that albumin induces tubular unfolded protein response...

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Proteinuria and hyperglycemia induce endoplasmic reticulum stress.

The endoplasmic reticulum (ER) is an important site for protein folding and becomes "stressed" when its capacity to fold proteins is overwhelmed. In response, "unfolded protein response" (UPR) genes are induced, increasing the capacity to fold proteins; if the response is insufficient, then apoptosis ensues. For investigation of whether proteinuria and hyperglycemia induce ER stress in renal ep...

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Endoplasmic reticulum stress inhibition attenuates hypertensive chronic kidney disease through reduction in proteinuria

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ژورنال

عنوان ژورنال: Nature Communications

سال: 2016

ISSN: 2041-1723

DOI: 10.1038/ncomms10330